参考文献

［1］ UMANATH K, LEWIS J B. Update on diabetic nephropathy: core curriculum 2018 [J]. Am J Kidney Dis, 2018, 71 (6): 884-895.

［2］ ANDERS H J, HUBER T B, ISERMANN B, et al. CKD in diabetes: diabetic kidney disease versus nondiabetic kidney disease [J]. Nat Rev Nephro, 2018, 14 (6): 361-377.

［3］ HIRANO T. Pathophysiology of diabetic dyslipidemia [J]. J Atheroscler Thromb, 2018, 25 (9): 771-782.

［4］ SANAJOU D, GHORBANI HAGHJO A, ARGANI H, et al. AGE-RAGE axis blockade in diabetic nephropathy: Current status and future directions [J]. Eur J Pharmacol, 2018, 833: 158-164.

［5］ TONNEIJCK L, MUSKIET M H, SMITS M M, et al. Glomerular hyperfiltration in diabetes: mechanisms, clinical significance, and treatment [J]. J Am Soc Nephrol, 2017, 28 (4): 1023-1039.

［6］ PREMARATNE E, VERMA S, EKINCI E I, et al. The impact of hyperfiltration on the diabetic kidney [J]. Diabetes Metab, 2015, 41 (1): 5-17.

［7］ THOMAS M C, CHERNEY D Z I. The actions of SGLT2 inhibitors on metabolism, renalfunction and blood pressure [J]. Diabetologial, 2018, 61 (10): 2098-2107.

［8］ SAAD A, HERRMANN S M, TEXTOR S C. Chronic renal ischemia in humans: can celltherapy repair the kidney in occlusive renovascular disease [J]？Physiology (Bethesda), 2015, 30 (3): 175-182.

［9］ LYTVYN Y, GODOY L C, SCHOLTES R A, et al. Mineralocorticoid antagonism and diabetic kidney diseasel [J]. Curr Diab Rep, 2019, 19 (1): 4.

［10］ JANKOVIC A, KORAC A, BUZADZIC B, et al. Targeting the NO/superoxide ratio in adipose tissue: relevance toobesity and diabetes management [J]. Br J Pharmacol, 2017, 174 (12): 1570-1590.

［11］ YUAN T, YANG T, CHEN H, et al. Newinsights into oxidative stress and inflammation during diabetesmellitus-accelerated atherosclerosis [J]. Redox Biol, 2019, 20: 247-260.

［12］ 剧孟磊, 左洋洋, 赖宇雄, 等. 线粒体活性氧—糖尿病肾病进展的中心环节 [J]. 中华肾脏病杂志, 2015, 31 (11): 866-869.

［13］ YANG Q, WU F R, WANG J N, et al. Nox4 in renal diseases: An update [J]. Free Radic Biol Med,2018, 124: 466-472.

［14］ PANDEY V K, MATHUR A, KAKKAR P. Emerging role of unfolded protein response (UPR) mediated proteotoxic apoptosis in diabetes [J]. Life Sci, 2019, 216: 246-258.

［15］ 左洋洋, 剧孟磊, 王惠珍, 等. 氧化低密度脂蛋白通过PTEN/SR-A途径导致足细胞损伤 [J]. 中华肾脏病杂志, 2015, 31 (10): 766-773.

［16］ 剧孟磊, 王慧珍, 左洋洋, 等. RhoA激酶1在氧化低密度脂蛋白诱导足细胞损伤中的作用 [J]. 中华肾脏病杂志, 2017, 33 (1): 37-42.

［17］ VERGÈS B. Pathophysiology of diabetic dyslipidaemia: where are we [J]?Diabetologia, 2015, 58 (5):886-899.

［18］ BEHL T, VELPANDIAN T, KOTWANI A. Role of altered coagulation-fibrinolyticsystem in the pathophysiology of diabetic retinopathy [J]. Vascul Pharmacol, 2017, 92: 1-5.